Vaccine Patrol
NPR said, “No major religion has come out in opposition to the COVID-19 vaccines. Jehovah’s Witnesses, Christian Scientists, and the Catholic Church have all issued statements saying that their religion does not prohibit members from receiving the COVID-19 vaccine. The Pope has declared getting vaccinated “an act of love.” And California is vaccinating every single child in the state without any science supporting such a drastic measure. Biden has blown twenty figures building an infrastructure of compliance with non-profits and a multitude of government enforcers who are certainly willing to implement whatever mandates they have in store for us next. Remember, this is just the beginning.
The claim is misinformation but no one mentions specifics. An open dialogue about these issues is way overdue.
https://youtu.be/3XAMPFNdUrs
Why recommend those who have antibodies from Covid get vaccinated?
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WHO HANDED THE KEYS TO FAUCI?!
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SARS-CoV-2 Spike Proteins and Cell-Cell Communication Inhibits TFPI and Induces Thrombogenic Factos in Human Lung Microvascular Endothelial Cell and Neutrophils: Implications for COVID-19 Coagulopathy Pathogenisis
In SARS-CoV-2-infected humans, disease progression is often associated with acute respiratory distress syndrome involving severe lung injury, coagulopathy, and thrombosis of the alveolar capillaries. The pathogenesis of these pulmonary complications in COVID-19 patients has not been elucidated. Autopsy study of these patients showed SARS-CoV-2 virions in pulmonary vessels and sequestrated leukocytes infiltrates associated with endotheliopathy and microvascular thrombosis. Since SARS-CoV-2 enters and infects target cells by binding its spike (S) protein to cellular angiotensin-converting enzyme 2 (ACE2), and there is evidence that vascular endothelial cells and neutrophils express ACE2, we investigated the effect of S-proteins and cell–cell communication on primary human lung microvascular endothelial cells (HLMEC) and neutrophils expression of thrombogenic factors and the potential mechanisms. Using S-proteins of two different SARS-CoV-2 variants (Wuhan and Delta), we demonstrate that exposure of HLMEC or neutrophils to S-proteins, co-culture of HLMEC exposed to S-proteins with non-exposed neutrophils, or co-culture of neutrophils exposed to S-proteins with non-exposed HLMEC induced transcriptional upregulation of tissue factor (TF), significantly increased the expression and secretion of factor (F)-V, thrombin, and fibrinogen and inhibited tissue factor pathway inhibitor (TFPI), the primary regulator of the extrinsic pathway of blood coagulation, in both cell types. Recombinant (r)TFPI and a thiol blocker (5,5′-dithio-bis-(2-nitrobenzoic acid)) prevented S-protein-induced expression and secretion of Factor-V, thrombin, and fibrinogen. Thrombomodulin blocked S-protein-induced expression and secretion of fibrinogen but had no effect on S-protein-induced expression of Factor-V or thrombin. These results suggests that following SARS-CoV-2 contact with the pulmonary endothelium or neutrophils and endothelial–neutrophil interactions, viral S-proteins induce coagulopathy via the TF pathway and mechanisms involving functional thiol groups. These findings suggest that using rTFPI and/or thiol-based drugs could be a viable therapeutic strategy against SARS-CoV-2-induced coagulopathy and thrombosis. View Full-Text
Oro-Systemic Health Symposium 2022 – Part 3: Graham Lloyd-Jones – The anatomy of COVID-19
https://youtu.be/pPsbO1Qw2GM Dr. Graham Lloyd-Jones, Consultant Radiologist at Salisbury NHS Foundation Trust and Director of Radiology Masterclass, describes the nature of...